- Myocardial infarction (MI)
- End-stage cardiomyopathy
- Papillary muscle or valve rupture
- Cardiac tamponade
- Pulmonary embolism (PE)
- Sudden, severe, extreme heart failure
- Decreased Perfusion
- ↓ CO
- ↓ BP
- ↑ HR (compensation)
- ↑ SVR (compensation)
- Weak, thready pulses
- Cool, diaphoretic skin
- Pale, dusky, cyanotic, or mottled skin
- ↓ urine output
- ↓ LOC, anxiety
- Volume Overload
- ↑ CVP
- Pulmonary Edema
- Pink, frothy sputum
- Sudden, severe SOB
- Muffled Heart Sounds
- S3, S4 present
- Therapeutic Management
- Treat Cause
- Revascularization for MI (PCI, CABG)
- Thrombolytics or surgical removal for PE
- Pericardiocentesis for cardiac tamponade
- Improve Contractility
- Dopamine – may ↑ HR
- Decrease Afterload
- Furosemide – for Pulmonary edema
- Caution – may ↓ BP
- Surgical Intervention
- Heart Transplant
- Treat Cause
- Patient Education
- Health promotion for prevention of myocardial infarction (see MI lesson)
- Explain procedures and expectations
- Symptoms to report to RN or HCP
- Importance of positioning (HOB > 30° and legs elevated)
- Medication instructions, side effects
Cornell Note-Taking System Instructions:
- Record: During the lecture, use the note-taking column to record the lecture using telegraphic sentences.
- Questions: As soon after class as possible, formulate questions based onthe notes in the right-hand column. Writing questions helps to clarifymeanings, reveal relationships, establish continuity, and strengthenmemory. Also, the writing of questions sets up a perfect stage for exam-studying later.
- Recite: Cover the note-taking column with a sheet of paper. Then, looking at the questions or cue-words in the question and cue column only, say aloud, in your own words, the answers to the questions, facts, or ideas indicated by the cue-words.
- Reflect: Reflect on the material by asking yourself questions, for example: “What’s the significance of these facts? What principle are they based on? How can I apply them? How do they fit in with what I already know? What’s beyond them?
- Review: Spend at least ten minutes every week reviewing all your previous notes. If you do, you’ll retain a great deal for current use, as well as, for the exam.
For more information, visit www.nursing.com/cornell
Okay guys, let’s talk about the basics of cardiogenic shock.
Remember the initial insult here is a broken or blocked pump. There are three main causes – myocardial infarction (remember dead heart muscle can’t pump), cardiac tamponade (which is fluid around the heart that compresses it and makes it hard to pump), and a pulmonary embolism that keeps blood from moving forward through the lungs. So the big thing we want you to see is that this is like an extreme version of heart failure. The blood can’t go forward so it backs up – so we have an increased preload. The bad pump causes our cardiac output and blood pressure to drop. The body tries to compensate by increasing the heart rate, and also by vasoconstricting and shunting blood to vital organs, so we see our afterload increase as well. What we’re left with is a pumping system that is entirely broken and vital organs that aren’t getting the blood they need. So what does this look like in our patient?
Well remember it’s like extreme, sudden, severe heart failure – so you’ve lost all perfusion forward. What happens with a lack of perfusion to the brain? Decreased LOC. Decreased perfusion to the kidneys? Decreased urine output. Decreased perfusion to the skin and extremities? Cold, pale, clammy skin, decreased pulses, slow cap refill. ALL signs of decreased perfusion. That’s your #1 clue. We may also see the problems of the blood backing up, right? Remember what happens when the blood backs up into the lungs – we get pulmonary edema and hear crackles. Then when it starts backing up into the head we see that extreme JVD – like a rope in their neck, right? So can you see how this is just sudden, extreme, severe heart failure? So keep that in mind as we move forward.
When we start thinking about treating a patient in cardiogenic shock, the most important thing is to identify and treat the cause. If they’ve had an MI, they’ll need revascularization like we talked about in the MI lesson. If it’s cardiac tamponade we need to remove the fluid from around their heart, if it’s a pulmonary embolism, we need to bust up or remove that clot. If we don’t address the cause, nothing else we do will be effective. When it comes to medication management, the big goal is to get the heart pumping more effectively and decrease the pressure it has to pump against. The top two drugs we give for patients in cardiogenic shock are dopamine and dobutamine. They will both increase contractility. Dopamine can also increase heart rate, while dobutamine can also help with vasodilation to decrease afterload. Patients who have had an MI will also still get the standard MONA treatment as well. If the patient has developed severe pulmonary edema, they may also receive a diuretic like Furosemide to offload that volume. We just have to be careful not to drop their blood pressure too much.
So, there are a lot of nursing priorities for this patient – one of which being that they need to be in an ICU. But we’re gonna focus on the top 3 concepts here. If you check out the outline and the care plan attached to this lesson, you’ll see a ton of details about specific interventions. The first concept is perfusion, we have got to monitor their hemodynamics and maintain a good cardiac output. Then, because these patients are at risk for airway and breathing issues, we need to monitor their oxygen status and intervene as needed. Then finally I added clotting because this condition might be caused by a clot in the coronary arteries or pulmonary arteries or might be because of bleeding around the heart – so we need to consider the interventions required to manage those conditions as well.
So let’s recap – cardiogenic shock is caused by a broken or blocked pump – that might be an MI, cardiac tamponade, or a PE. Most of the symptoms you see are caused by a lack of perfusion to the organs like the brain, kidneys, and skin, and by the backup of blood into the lungs and body (that’s why you see pulmonary edema and JVD). Remember it’s like a sudden, extreme version of heart failure. Treatment is focused on treating the cause, increasing contractility and getting the heart pumping more efficiently against less pressure. Our priorities are going to be perfusion and oxygenation, and then dealing with any clotting or bleeding issues depending on the cause. And finally, remember that this is an emergency, these patients need to be in an ICU and may even need to be on life support. So don’t be afraid to ask for help if you need it!
Make sure you check out the care plan and outline in this lesson to see lots of details about nursing care and interventions. We love you guys! Happy nursing!